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While the exact mechanism is not fully understood, GSTO1 appears to be associated with the early responses in the pro-inflammatory pathway, influencing oxidative and metabolic changes 43

GHK-Cu may influence (which I did not expect): Transcriptomics: Over 4,000 human genes modulated in studiesmany tied to wound healing, antioxidant defense, and anti-fibrosis

Another critical enzyme is fructose-1,6-bisphosphatase 1 (FBP1), a rate-limiting enzyme in gluconeogenesis that is markedly downregulated in CRC [98]