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iron, thrombin, glutamate) directly induces neuronal hyperexcitability and network reorganization [71, 72], while albumin (Alb) leakage triggers TGF-mediated astrocytic activation, impairing potassium buffering and promoting excitatory synaptogenesis [73, 74]

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Hemodynamic and histopathological changes in the early phase of the development of an intracranial aneurysm