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The results of these experiments have been favorable, with benefits speculated to arise from metabolic modulation, cardiomyocyte apoptotic inhibition, reduced cardiac inflammation, and augmented endothelial function

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were previously employed by Novo Nordisk

The drug was originally approved by the FDA in 1979 as an antihypertensive (brand name Loniten) at doses of 10 to 40 mg daily [1]

Therefore, this review summarizes the mechanisms of ferroptosis, its functional impacts on wound repair-related cells (macrophages, fibroblasts, endothelial cells, keratinocytes) and wound bacterial infections

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