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As NO levels increase, PTP activity is partly restored, counteracting the inhibitory influence of ROS and reducing the hypoxia-driven rise in tyrosine phosphorylation on Band 3

Mechanistic endpoints were recorded when available, including markers of inflammation, angiogenic activity, collagen organization, and bone remodeling

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Gamma-glutamyl transpeptidase-mediated reactions are known to be catalyzed by nucleophilic attack on the donor substrate