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Sequence analysis revealed a significantly higher divergence from the gt1b consensus for the ReED of FCH patients than for non-FCH patients, which was only driven by accumulation of mutations in the ISDR but not in the C-terminal part (C-term) of the ReED (Fig
The administration of riboflavin in animal models of chronic degenerative diseases associated with ROS, such as diabetes, renal toxicity, and hepatotoxicity [61], resulted in elevated levels of antioxidant enzymes, including glutathione reductase, and reduced glutathione, indicating that this vitamin has an impact on the antioxidant status
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