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Tissue fibroblasts in each species, with different types of placentation, have evolved to respond to invasive cancer cells similarly to how their endometrial fibroblasts react to placental invasion (147)

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Compared to BPC-157, which acts primarily via the nitric oxide and growth hormone receptor pathways, and TB-500, which functions through actin sequestration and VEGF signalling, GHK-Cu operates largely through copper-mediated gene expression modulation and extracellular matrix remodelling

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