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Although MCs have long been recognized to shape disease by tuning mitochondrial metabolism, their role in oncogenesis has come into focus only within the past decade [9]

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However, human clinical trials remain scarce

The activation of NRF2 turns on several classical anti-ferroptotic genes, including GPX4 and ferritin heavy chain 1 ( FTH1 )

They started looking at what FOXO4 actually does in senescent cells, and one important thing was that it liked to grab on (bind) to a protein called p53