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Perhaps not surprising, this hindbrain site of action is also responsible for mediating the illness-like behaviors (e.g., nausea, conditioned taste avoidance, emesis) of systemically delivered GLP-1R agonists (43)

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This effect is of critical importance for CV risk management in T2D, given the high prevalence of hypertension and its well-established role in driving CV event rates within this population [62]

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