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This secondary binding site may underlie some of the peptide's reported cytoprotective and anti-inflammatory properties observed in preclinical models, though the clinical significance remains under investigation

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349 Aging-related nuclear NAD + decline inhibits the mitochondrially encoded genes via the SIRT1-HIF-1-c-Myc pathway, while boosting NAD + levels rescues the mitochondrial function in old mice in a SIRT1-dependent manner

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