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Specifically, ferroptosis acts as a critical mediator of this epithelial cell death, thereby exacerbating barrier dysfunction (Chen et al., 2021)

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Furthermore, the virulence was higher in air polluted areas, with adverse effects on respiratory diseases (204)

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Given its regenerative properties and favorable safety profile, AOD9604 holds promise as a novel therapeutic agent for OA, especially when used in conjunction with established treatments like HA [1]

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Therefore, we hypothesized that silencing this response upon refeeding plays a more significant role in longevity by restoring and preserving long-term lipid homeostasis

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Immune peptides function as modulators, steering inflammation either up or down as necessary

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