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In both cases the 2 -adrenoceptor-dependent current was not reduced by opioid exposure (Bailey et al., 2004, 2009a,b) indicating no loss of the ability of the effector ion channel (GIRK) to signal

Similar in vitro work showed that HGF promoted dendritic arborization in hippocampal neurons, an effect that could be blocked by NMDA receptor antagonists and was dependent on intracellular calcium and CaMKII activity [2]

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Risk increases significantly in individuals with copper metabolism disorders (Wilsons Disease)