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NLY01 exerts its neuroprotective effect by potently suppressing this pathogenic inflammatory axis, thereby attenuating the formation of cytotoxic A1 astrocytes and preserving RGC viability [42]

However, newer evidence suggests the relationship is more complex and that GIP activation in the context of GLP-1 activation might actually support fat burning

A study by Mitchell et al used a surgically implanted intracranial pressure (ICP) monitor and showed reduction in ICP within minutes to hours after GLP-1 RA initiation long before weight loss would have occurred

Findings reported in the New England Journal of Medicine and through Eli Lillys international research programme confirm its potential as a strong oral alternative to injectable GLP-1 therapies

This is underscored by the results of a clinical trial comparing the development of obese adults who received either semaglutide or liraglutide in addition to nutritional advice and physical activity, and significantly greater weight loss was achieved in the semaglutide group [102]