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This led to a reduction in the expression of the fibrosis genes actin-alpha-2 (ACTA2) and collagen-type-1-alpha-1 (COL1A1) in the kidneys, suggesting that the inhibitor may have therapeutic promise for treating renal fibrosis[83]

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In addition, GHK-Cu influences cellular proliferation, migration, and differentiation, supporting angiogenesis and stem cell function, ultimately promoting tissue repair and remodeling in diverse tissues including skin, bone, and lung 1-4