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Pentoxifylline modulates p47phox activation and downregulates neutrophil oxidative burst through PKA-dependent and -independent mechanisms
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Therefore, GSH degradation (but not GSH per se) contributes to cysteine pool which regulates mTORC1 pathway and the ISR in HepG2 cells
When activated, macrophages can produce high levels of nitric oxide through inducible nitric oxide synthase
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