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This chronic bile acid exposure increases colonic irritation and diarrheaboth risks compounded by GLP-1 medications

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This effect has been noted with GLP-1 receptor agonists as a class and may be further influenced by glucagon receptor agonism

The body's glucose-sensing mechanisms, which have adapted to chronically elevated blood sugar levels, may initially interpret normal or improved glucose levels as relatively low, potentially triggering hunger signals and cravings for quick-acting carbohydrates