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Likewise, infiltration of B cells into synovial tissue leads to autoantibody production against citrullinated peptides (ACPAs) and RF that can amplify the production of proinflammatory cytokines through synovial macrophages and subsequently the production of ROS [3,131]

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By inhibiting TRPS1 and thereby elevating ZEB2 levels, miR-221/222 reduce E-cadherin expression and promote EMT, contributing to the aggressive phenotype of basal-like breast tumors [64, 65]

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