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Senescent fibroblasts in diabetic wounds exhibit ferroptosis resistance due to defective ferritinophagy, but upregulating the expression of the ferritin autophagy-related molecule NCOA4 increases their susceptibility to ferroptosis and improves wound healing [81]

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Spiering BA, Kraemer WJ, Vingren JL, Hatfield DL, Fragala MS, Ho J-Y, Maresh CM, Anderson JM, Volek JS

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