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The initial entry of the virus into human cells was found to be via the SARS-CoV-2 spike (S) protein binding to the cell surface receptor angiotensin-converting-enzyme-2 (ACE2) expressed by type II pneumocytes [1]

meningitidis Within a genus such as Neisseria , characterized by notable genomic plasticity and consequent genetic heterogeneity, a key point of concern is the emergence of species potentially pathogenic for humans and other animals

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