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Moreover, both coimmunoprecipitation and confocal microscopy studies showed that PMA promoted the interaction between cPKC and GSTP in LPS-unstimulated cells, while GF109203X inhibited the binding of cPKC to GSTP in LPS-stimulated cells (Figures 6D,E)

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The amylin pathway is distinct from both GLP-1 and GIP signaling

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