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The resulting no-reflow phenomenonwherein tissue perfusion remains inadequate despite macrovascular restorationexacerbates hypoxia and creates a self-amplifying cycle of inflammation, mitochondrial damage, and microcirculatory failure (95)
9 Tissue response to amalgam depends on the particle size and composition, and a foreign-body reaction with a mononuclear inflammatory cell infiltrate is often noted
DNA methylation cannot distinguish the molecular class (Figure 4)
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Mitochondrial etiology of neuropsychiatric disorders
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