Mitochondrial GPx1 decreases induced but not basal oxidative damage to mtDNA in T47D cells
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If left untreated, a B12 deficiency can cause more severe health problems, including nerve damage, cognitive issues, and anaemia
Through a system involving the enzymes carnitine palmitoyltransferase I (CPT-I) and carnitine palmitoyltransferase II (CPT-II), L-carnitine binds to fatty acid molecules, transports them into the mitochondrial matrix, and releases them for beta-oxidation, the process that breaks fatty acids down into acetyl-CoA units for energy production in the citric acid cycle
Minimize transit time
Fe 3+ is released from LCN2 and may be reduced to Fe 2+ and transported to the cytoplasm, enabling efficient iron recycling by the cell