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doi: 10.1016/S0140-6736(21)01751-7

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suggest that GHK-Cu may also tone down inflammatory signaling in macrophages activated by pro-inflammatory triggers and in lung cell injury models

LPS administration induced significant phosphorylation of NF-B p65 (Ser536), p38 MAPK, and JNK1/2 in lung homogenates

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