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By suppressing M1 polarization and promoting the transition to the anti-inflammatory M2 phenotype, this intervention improved the inflammatory microenvironment, accelerated re-epithelialization and collagen deposition, and enhanced healing of diabetic and infected wounds

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Intestinal epithelial cells (IECs) also express transmembrane mucins, such as MUC12, MUC1, and MUC3 attached to the apical surface and forming, together with the glycolipids, the glycocalyx [5]

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