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BPC-157 has been investigated for interactions with VEGFR2-, ERK1/2-, eNOS-, and FAK-associated signaling networks together with regulatory processes involving extracellular protein systems

Erratum in: J Hepatol

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The neuroprotective mechanisms may involve modulation of inflammatory responses within the central nervous system, promotion of neuronal survival, and enhancement of axonal regeneration and synaptic plasticity

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Its clinical development for obesity was discontinued in 2007 after the pivotal 536-participant Phase IIb trial failed to meet its primary efficacy endpoint, and no formulation of the compound has received FDA approval since