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Kallistatin/Serpina3c inhibits cardiac fibrosis after myocardial infarction by regulating glycolysis via Nr4a1 activation

10.1161/01.hyp.25.5.924 28 DemaegdtH.LaeremansH.De BackerJ.-P.MosselmansS.LeM

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Increasing the dose more slowly than usual, or 3

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TLR signaling molecules, like mitogen-activated protein kinases (MAPK) and phosphoinositide 3-kinase (PI3K)/protein kinase B (Akt), play fundamental roles in TLR-mediated cell proliferation and survival via reducing apoptosis and increasing time for viral replication (Li et al., 2010