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The pathophysiology of AKI in sepsis is complex and multifactorial, encompassing intrarenal hemodynamic changes, endothelial dysfunction, infiltration of inflammatory cells within the renal parenchyma, and obstruction of renal tubules by necrotic cells and debris (112,113)

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This exclusion is not optional and is screened for during evaluation

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With the major changes in dietary macronutrient composition that occur in the transition to a KD, there may be substantial changes in the environment for gut microbes