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, Genetic evidence for repurposing of GLP1R (Glucagon-like peptide-1 receptor) agonists to prevent heart failure, J Am Heart Assoc, vol

Closely associated with complex metabolic disorders, NASH progresses through chronic inflammatory processes that promote and sustain a pro-oncogenic environment, leading to hepatocyte injury, thereby emerging as a principal etiological factor of terminal liver diseases, including hepatocellular carcinoma (HCC) and liver failure [2, 3]

Possible therapeutic combinations of agents may be suggested by trial outcomes

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Weber, H.E