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To examine the phosphorylation capacities of c-MET mutants, 3UTR siRNA (hs.Ri.MET.13.1 from IDT) was used for selective knockdown of endogenous c-MET in H1299 cell lines that stably express near-endogenous myc-c-MET (WT or mutant)

However, the overall side effect profile in clinical trials was generally comparable to tirzepatide, with gastrointestinal events being the most common and dose-limiting factor

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Beyond the predominant 2D form, GO exists in several other dimensionalities that offer significant advantages