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Taken together, these pharmacokinetic findings indicate that the centrally mediated effects of GLP-1RAs are likely due to limited brain penetration or action at sites adjacent to the BBB, rather than widespread distribution throughout the central nervous system

(3) limited bioavailability of selenium depending on its source and form

GLP-1RAs, such as liraglutide, inhibit the PINK1/Parkin pathway and restrain excessive mitophagy, protecting RGCs from a high glucose environment

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