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Janus kinase inhibitors (JAKi) including tofacitinib and baricitinib modulate SLE pathogenesis via JAK/STAT pathway inhibition, with preclinical evidence revealing tofacitinibs dual capacity to reduce NET burden and restore endothelial homeostasis in MRL/lpr models (148150)

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The medication works by mimicking the naturally occurring GLP-1 hormone, which enhances glucose-dependent insulin secretion, suppresses glucagon release, slows gastric emptying, and reduces appetite

Curcumin prevents Cr(VI)-induced renal oxidant damage by a mitochondrial pathway