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In addition, in several studies, NAD depletion enhanced cancer cell susceptibility to oxidative stress through a reduction in antioxidative capacity by downregulating antioxidant proteins

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These effects were associated with increased F-actin formation and activation of focal adhesion kinase (FAK) and paxillin phosphorylation, signaling events central to cell attachment and cytoskeletal remodeling during tissue repair processes [4]

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