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They noted a negative correlation between GPX3 expression levels and fibrosis levels across various treatment groups of the bromodomain inhibitor JQ1, suggesting a potential role of GPX3 in promoting the differentiation of cardiac fibroblasts into a protective state

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doi: 10.1002/mnfr.200700050 121

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Disruptions in this axis, driven by gut dysbiosis, have been implicated in the pathogenesis of a wide range of neurodegenerative and neuropsychiatric disorders