In an in vitro model of diabetes retinopathy, AC, reduced retinal pigment epithelial cell damage through inhibiting Keap1 expression, endorsing nuclear translocation of Nrf2, elevating NQO1 and HO-1 expression, reducing ROS, and increasing superoxide dismutase (SOD), total antioxidant capacity (T-AOC), and glutathione peroxidase (GSH-Px) (Yang et al
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Unlike testosterone therapy, estrogen therapy, growth hormone therapy, or other hormone-based treatments, BPC-157 is generally not considered a hormone replacement treatment
This pharmacokinetic limitation poses a significant challenge for its therapeutic application, as only a small fraction of orally administered AC reaches systemic circulation, reducing its efficacy in vivo
It is important to note that while transdermal delivery of glutathione offers potential benefits, the effectiveness of this route of administration and its impact on specific disease states require further clinical research
