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Pharmacological modulation of mitochondrial calcium uniporter controls lung inflammation in cystic fibrosis

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TB-500 operates systemically as a **cytoskeleton remodeler**: its primary function is G-actin monomer sequestration (active N-terminal Ac-Ser-Asp-Lys-Pro), its cleaved AcSDKP sequence is an **autonomous angiogenic factor**, it upregulates laminin-5 and matrix metalloproteinases (MMPs), and mobilizes mesenchymal progenitor cells

Detoxifies chemicals: In the liver, glutathione binds to toxins, making them easier to excrete

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