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Studies further elucidate pathological interactions between the mitochondrial fission regulator Drp1, phosphorylated tau (p-tau), and A

Fermented vs

doi:10.1089/jmf.2007.0075

Basat S, Sivritepe R, Ortaboz D, Sevimalk E, Kk EV, et al

The improvements includes oxidative stress, glutathione deficiency and multiple aging hallmarks affecting mitochondrial dysfunction, mitophagy, inflammation, insulin resistance, endothelial dysfunction, genomic damage, stem cell fatigue and cellular senescence

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