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Patients with homozygosity at this mutated locus tend to exhibit a more severe clinical phenotype, usually associated with severe mental retardation and early onset seizures, and five of them died early in life [6,7,8]

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There is no evidence that it is mutagenic or carcinogenic in the conventional sense

This is part of why understanding how peptides work at the receptor level gives you a more nuanced perspective on timing

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