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As expected, H 2 O 2 (0.2 mM) exposure disrupted cellular glutathione homeostasis in both HepG2 cells (a human hepatoma cell line) and NCTC1469 cells (a non-transformed mouse hepatocyte cell line), as demonstrated by an increase in the GSSG/GSH ratio over time (Fig
A study exploring the response of Atgstu19 and Atgstu24 mutants to TuMV infection showed significant differences in specific AtGSTU gene expression, virus concentration, ultrastructural alterations, glutathione content, and glutathione transferase and reductase activities compared with Col-0 (wild-type) and mock-inoculated plants ( Atgstu24 mutants had a resistance-like reaction to TuMV (with a high decrease in virus gene expression and movement) compared to susceptible Col-0 plants, suggesting that GSTU24 may suppress plant resistance
Biochemical parameters for liver, kidney and heart as well as histological analysis showed no injury caused by the CD5/ LNP-FAPCAR 2.2 treatment (Table S3, S4 and Fig
Sulforaphane Augments Glutathione and Influences Brain Metabolites in Human Subjects: A Clinical Pilot Study Park, S., et al
Bibcode:1990FEBSL.267..189A