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Due to the exhaustion of cellular and extracellular GSH caused by numerous GSH-consuming pathways the severe inflammation and oxidative stress triggered by the viral infection steals GSH from crucial functions like NO-dependent vasodilatation, disallowing the patient of being protected from an inflammation that can become fatal

Programmed cell death protein 1 (PD-1) and its ligand PD-L1 are common targets for cancer immunotherapy and interact with a low intrinsic affinity of around 8 M 24

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