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The proposed mechanism most often cited involves GIP-receptor-mediated effects on adipose insulin sensitivity and possibly on central energy balance, though attribution between the two receptors in vivo remains an area of ongoing study

The blood sugar improvements are substantial and clinically meaningful

These effects may be connected to modulation of growth hormone and IGF-1 signaling, as well as alterations in hepatic urea cycle enzyme activity

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Sensors and Actuators B: Chemical, 242 , 679686

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