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Combined intestinal loss of PPAR// attenuates KD-driven intestinal stem cell expansion, proliferation and clonogenicity, whereas inhibition of downstream fatty acid oxidation through CPT1A loss limits adenoma formation specifically under KD, linking tumour initiation to fatty acid oxidation of dietary lipids rather than lipid accumulation

DOI: 10.1002/ana.410420504 23

Consequently, low activation prevented proliferation in vitro (Supplementary Fig

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1 These frequent respiratory interruptions result in periods of low blood oxygen levels, nervous system dysfunction, and sleep fragmentation