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Activated astrocytes can shift toward different phenotypes: the A1 type, induced via NF-B signaling, produces proinflammatory mediators and can trigger neuronal apoptosis, whereas the A2 phenotype, driven by STAT3 activation, promotes anti-inflammatory and neuroprotective responses [32]

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Although BMSCs combined with calcium alginate composite gel injection therapy [146]and tissue-engineered slings incorporating BMSCs with biodegradable silk fabric scaffolds [147] have shown potential in treating SUI, their clinical applications have yet to be validated

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Fenfluramine and dexfenfluramine were withdrawn because of heart valve damage