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1 PROGRESSION TO TYPE 2 DIABETES # The rate of progression to diabetes in patients without diabetes at baseline was also studied as part of the trial

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Results Here we show that in vivo genome editing enables sustained Exendin-4 secretion from liver cells, leading to prolonged elevation of Exendin-4 levels in the bloodstream

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The proposed mechanism involves PCs activating peroxisome proliferator-activated receptor (PPAR) in macrophages, upregulating PPAR target genes (CD36 and ABCG1) and driving the transition from M1 to M2 phenotypes

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