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10.1002/term.2914 14 BydonM.QuW.MoinuddinF

We propose that the increase in circulating glucagon observed following genetic/pharmacological inactivation of glucagon signalling in mice and in people with type 2 diabetes reflects the removal of GLP-1(936)s glucagonostatic action

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Finally, in our isogenic GBM models, we confirmed that overexpression of PSPH and PSPH V116I enhanced galectin-1 expression, measured by immunoblot (Figs

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Researchers utilized the Atropos Health Eos electronic health record dataset