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The development of FoB in B cell-specific Gclc -deficient mice can be linked to the metabolic quiescence which characterizes wild-type FoB 38

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Specifically, the ROS scavenger butylated hydroxyanisole delayed TNF-induced necroptosis but had no effect on necroptosis induced by direct RIPK3 oligomerization, suggesting that ROS may facilitate upstream signaling events rather than serve as an obligate execution mechanism 80