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While a rise in GSH appears to be a universal cellular response to oxidative stress, decreased GSH levels tend to develop some diseases like mitochondrial diseases (Enns et al

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More specifically, a complex regulation at the DNA level (transcriptional control), at the mRNA level (post-transcriptional control), and at the protein level (protein stability, availability of binding partners, and post-translational control) is responsible for maintaining cellular redox homeostasis (Fig

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