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found that these two vectors were highly expressed in myocardial mitochondria and showed that inhibition of DIC and OGC increased mitochondrial ROS and GSH depletion, exacerbating ferroptosis in the myocardium [60]

p53 mutation may promote hypoxia-induced genomic instability, leading to the activation of pro-oncogenic signaling such as hypoxia-inducible factor (HIF) 1 [4]

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At BUBS, our Collagen Peptides are hydrolyzed, which means the large protein molecules have been broken down into smaller, more easily absorbed chains called peptides

PET studies have detected glucose hypometabolism in the striatum, which often occurs years before the clinical symptoms [45]

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