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Therapeutic perturbation of the CoQ 10 redox axis therefore offers an attractive opportunity to sensitize tumors to ferroptotic cell death, particularly when combined with treatments that elevate oxidative stress or lipid peroxidation

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Frontiers in Nutrition, 6 , 146

FIGURE 3 The immunopharmacological effects of GSH are evident through several distinct but interconnected pathways, including regulation of inflammatory signaling networks like MAPK and NF-B, buffering of pathologic ROS as well as maintenance of cellular ROS signaling, and modulation of both peripheral and central immune cell activity (Figure 4)

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