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10.1136/bmjopen-2023-073275 403 SlykermanR
Mitochondria, the central organelles for cellular energy metabolism and oxidative stress regulation, serve a key role in ferroptosis (7)
These findings clearly show that GPX1 is irreplaceable by other selenoproteins in protecting against systemic oxidative stress, and GPX1 has a significant antioxidant function in the body (Liu et al., 2021)
During a copper-deficient state, the copper deficiency leads to mitochondrial dysfunction and ROS accumulation, which in-turn lead to cardiovascular diseases including myocardial I/R injury (94)
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