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Key points The pathological basis of interstitial cystitis/bladder pain syndrome (IC/BPS) remains unclear, with several hypotheses suggesting potential mechanisms, including autoimmune inflammation, oxidative stress and urothelial dysfunction

Apparently, no other signaling system is able to compensate for a lack of the c-Met in this process

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This FDA-approved methodology penetrates tissues, organs, and bones, to reactivate electrochemical function, regenerate nerve fibers, and enhance motor control

This represents a dramatic acceleration of surface healing