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There is abundant evidence that alterations in key neurotransmitters can both be modified by and contribute to oxidative stress and membrane dysfunction ( Figure 1 ), suggesting a link between these pathophysiological processes in SZ
In PK15 cells, while PCV2 replication appeared to be significantly induced by 0.1 M of OTA, SeMet, at concentrations of 2, 4, or 6 M, inhibited the viral replication process
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